β2‐Adrenergic stimulation enhances Ca2+ release and contractile properties of skeletal muscles, and counteracts exercise‐induced reductions in Na+–K+‐ATPase Vmax in trained men

It is shown that β2‐adrenergic stimulation counteracts exercise‐induced reductions in Na+–K+‐ATPase Vmax (maximum rate of activity) and elevates glycolytic activity during high intensity exercise and is important for the understanding of the role of β2-adreceptor activation in regulation of ion handling and contractile properties of non‐fatigued and fatigued skeletal muscles in humans.